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Transgenic approaches to defining the role of the stress-activated protein kinases, JNK and p38 in tumour progression
Reference
BBS/E/B/0000H457
Principal Investigator / Supervisor
Dr Simon Cook
Co-Investigators /
Co-Supervisors
Institution
Babraham Institute
Department
Babraham Institute Department
Funding type
Research
Value (£)
299,041
Status
Completed
Type
Institute Project
Start date
01/04/2003
End date
30/06/2006
Duration
39 months
Abstract
Many of the drugs commonly used in cancer chemotherapy activate the stress activated protein kinases signalling pathways. These pathways convey messages within the cell which can determine whether a cell lives or dies and so could influence the outcome of chemotherapy. Accordingly we need to know if activation of these signalling pathways is a desirable consequence of chemotherapy. This project will address this in tissue culture cell and transgenic mice models.
Summary
unavailable
Committee
Closed Committee - Biochemistry & Cell Biology (BCB)
Research Topics
X – not assigned to a current Research Topic
Research Priority
X – Research Priority information not available
Research Initiative
X - not in an Initiative
Funding Scheme
X – not Funded via a specific Funding Scheme
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