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Molecular basis of Th subset bias in intestinal nematode infections

ReferenceS06692
Principal Investigator / Supervisor Professor Richard Michael Maizels
Co-Investigators /
Co-Supervisors
Institution University of Edinburgh
DepartmentInst of Cell, Animal and Population Biol
Funding typeResearch
Value (£) 202,632
StatusCompleted
TypeResearch Grant
Start date 01/04/1997
End date 01/04/2000
Duration36 months

Abstract

Parasitic helminths are potent drivers of the T-helper type 2 (Th2) subset of lymphocytes. Th2 cytokines generate the exaggerated IgE and eosinophil responses observed in all animal host species infected with nematodes. We plan to identify novel mediators secreted by the rodent parasite Nippostrongylus brasiliensis which activate the Th2 pathway, and to define the primary host cells responding to these molecules. Secreted products will be characterised structurally and tested individually for immunological effects. These studies may identify a natural Th2 adjuvant of broad significance to immunology and animal health.

Summary

unavailable
Committee Closed Committee - Animal Sciences (AS)
Research TopicsX – not assigned to a current Research Topic
Research PriorityX – Research Priority information not available
Research Initiative X - not in an Initiative
Funding SchemeX – not Funded via a specific Funding Scheme
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