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The molecular and biochemical basis of cytoplasmic male sterility in sunflower
Reference
P12840
Principal Investigator / Supervisor
Professor Chris Leaver
Co-Investigators /
Co-Supervisors
Dr Janneke Balk
Institution
University of Oxford
Department
Plant Sciences
Funding type
Research
Value (£)
204,008
Status
Completed
Type
Research Grant
Start date
04/09/2000
End date
03/12/2003
Duration
39 months
Abstract
The expression of a mitochondrially encoded novel open reading frame appears to be causally related to the cytoplasmic male sterile (CMS) phenotype in most plants systems. However, how these novel protein(s) cause disruption of pollen development is unknown. Based on our preliminary data, we have proposed a hypothesis which suggests that, in sunflower, mitochondrial dysfunction is caused by the novel CMS specific ORF522 protein which is associated with the F1-F0-ATPase complex. We propose to investigate mitochondrial function in fertile, sterile and restored hybrid sunflowers, focussing on the assembly and function of the F1-F0-ATPase complex and the possible role of mitochondria in programmed cell death during male flower development.
Summary
unavailable
Committee
Closed Committee - Plant & Microbial Sciences (PMS)
Research Topics
X – not assigned to a current Research Topic
Research Priority
X – Research Priority information not available
Research Initiative
X - not in an Initiative
Funding Scheme
X – not Funded via a specific Funding Scheme
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