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Investigating if SI-induced actin disruption is involved in programmed cell death

ReferenceC18649
Principal Investigator / Supervisor Professor Vernonica E. (Noni) Franklin-Tong
Co-Investigators /
Co-Supervisors
Institution University of Birmingham
DepartmentSch of Biosciences
Funding typeResearch
Value (£) 217,288
StatusCompleted
TypeResearch Grant
Start date 09/08/2003
End date 08/08/2006
Duration36 months

Abstract

We have two separate events identified as being triggered in incompatible pollen: (1) programmed cell death and (2) sustained actin depolymerisation. The data suggest that these events may be linked. Animal cell studies suggest that F-actin depolymerisation acts as an executor of apoptosis. The aim of this proposal is to ascertain if this is the case; i.e. Is the actin depolymerisation and PCD linked by a common signalling cascade? Briefly, we wish to answer these interlinked questions: (A) Is F-actin depolymerisation an effector and executor of PCD in pollen? (i.e. is this required for PCD to initiate and or progress?) (B) Are caspase-like modification (e.g. cleavage) of actin and ABPs triggered? (i.e. are these actin cytoskeleton-based targets for PCD?).

Summary

unavailable
Committee Closed Committee - Biochemistry & Cell Biology (BCB)
Research TopicsX – not assigned to a current Research Topic
Research PriorityX – Research Priority information not available
Research Initiative X - not in an Initiative
Funding SchemeX – not Funded via a specific Funding Scheme
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