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Proteolipid protein gene dosage and myelination

ReferenceC18627
Principal Investigator / Supervisor Professor Ian Griffiths
Co-Investigators /
Co-Supervisors
Dr Thomas Anderson, Dr Mark McLaughlin
Institution University of Glasgow
DepartmentVeterinary School
Funding typeResearch
Value (£) 214,760
StatusCompleted
TypeResearch Grant
Start date 21/06/2003
End date 20/06/2006
Duration36 months

Abstract

Increased dosage of the myelin proteolipid protein (Plp) gene disrupts the co-ordinate programme of myelin gene expression and leads to dysmyelination or demyelination. The increased expression of the Plp gene does, in some circumstances, result in changes in levels of other proteins within the myelin sheath, suggesting local regulatory mechanisms. Morphological evidence suggests that excess PLP is degraded via the lysosomal system. This study examines the dynamics of PLP processing in the presence of increased gene dosage and the effect of this upon other key myelin proteins. The study provides a model in which to explore the complex gene interactions necessary to generate the myelin sheath in a highly specialised cell type.

Summary

unavailable
Committee Closed Committee - Biochemistry & Cell Biology (BCB)
Research TopicsX – not assigned to a current Research Topic
Research PriorityX – Research Priority information not available
Research Initiative X - not in an Initiative
Funding SchemeX – not Funded via a specific Funding Scheme
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