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Regulation of the size of the inositol trisphosphate-sensitive calcium stores by parathyroid hormone

ReferenceC13743
Principal Investigator / Supervisor Professor Colin Taylor
Co-Investigators /
Co-Supervisors
Institution University of Cambridge
DepartmentPharmacology
Funding typeResearch
Value (£) 170,368
StatusCompleted
TypeResearch Grant
Start date 01/11/2000
End date 31/03/2004
Duration41 months

Abstract

The parathyroid hormone receptor (PTHR1) belongs to a family of receptors that both stimulate cAMP formation and release intracellular Ca stores. The latter often requires neither cAMP-dependent protein kinase (PKA) nor formation of IP3. Our studies of cells transfected with PTHR1 suggest that PTH may be the first example of a hormone controlling the size of the IP3-sensitive Ca stores and so the amplitude and spatial distribution of IP3-evoked Ca signals. We suggest that PTH (and related receptors) stimulate fusion of IP3-sensitive and insensitive Ca stores by a PKA-independent mechanism that probably involves monomeric G proteins. We aim to define the signal linking PTH to Ca signals and establish whether it does cause fusion of Ca stores.

Summary

unavailable
Committee Closed Committee - Biochemistry & Cell Biology (BCB)
Research TopicsX – not assigned to a current Research Topic
Research PriorityX – Research Priority information not available
Research Initiative X - not in an Initiative
Funding SchemeX – not Funded via a specific Funding Scheme
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