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Development and application of an antisense based system to study the biological function of PrP in the hippocampus

ReferenceBS410546
Principal Investigator / Supervisor Dr Nikki Macleod
Co-Investigators /
Co-Supervisors
Dr Peter Estibeiro
Institution University of Edinburgh
DepartmentBiomedical Sciences
Funding typeResearch
Value (£) 135,274
StatusCompleted
TypeResearch Grant
Start date 01/06/1999
End date 01/12/2000
Duration18 months

Abstract

Mice lacking a functional prn-p gene do not develop spongiform encephalopathy when challenged with scrapie-infected material, indicating an important role for the gene product. Yet, PrP knockout mice have failed to yield a consistent phenotype. Possible reasons for this surprising result may be incomplete functional knockout of the gene product or the compensatory up- or down-regulation of other genes. A strategy that may be more revealing than germ line knockout might be the acute knockdown of gene expression through the use of antisense sequences. Organotypic hippocampal cultures will be exposed to PrP antisense sequences and the consequences of rapid PrP depletion on normal cell functions, such as copper binding, cell metabolism, membrane properties, pre- and post-synaptic mechanisms, synaptic plasticity and protein phosphorylation will be assayed by electrophysiological and biochemical means.

Summary

unavailable
Committee Closed Committee - Agri-food (AF)
Research TopicsX – not assigned to a current Research Topic
Research PriorityX – Research Priority information not available
Research Initiative Biology of Spongiform Encephalopathies - Phase 4 (BS4) [1998]
Funding SchemeX – not Funded via a specific Funding Scheme
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