Award details

Role of VRN5/VIN3 in the vernalization response of Arabidopsis

Reference	BBS/E/J/0000A233
Principal Investigator / Supervisor	Professor Dame Caroline Dean
Co-Investigators / Co-Supervisors
Institution	John Innes Centre
Department	John Innes Centre Department
Funding type	Research
Value (£)	173,695
Status	Completed
Type	Institute Project
Start date	01/02/2005
End date	31/07/2008
Duration	42 months

Abstract

The Dean laboratory has been identifying mutants defective in vernalization for several years and has cloned several of the genes important for mediating vernalization. A key property of vernalization is the ability of the plants to 'remember' that they have experienced winter. The work in the Dean laboratory has shown that this memory is controlled by changes in the proteins (histones) associated with the DNA of a particular gene (FLC) encoding a repressor of flowering. These changes regulate the gene in a so-called 'epigenetic manner' that is they lock the expression state of the gene into the 'off state' so in the absence of the cold signal the gene still stays off. Two of the recently cloned VRN genes (VIN3/VRN7 and VRN5) encode proteins with domains that might act as receptors for signalling molecules. This grant will analyse how the proteins from these two genes work together in the plant to control levels of the important floral repressor, FLC and mediate the vernalization response. This work will provide information on the regulation of genes in response to environmental signals that may be generally relevant to both plant and animal biologists and provide information and materials to potentially manipulate flowering time and vernalization response in a range of crops.

Summary

unavailable

Committee	Closed Committee - Genes & Developmental Biology (GDB)
Research Topics	Microbiology, Plant Science
Research Priority	X – Research Priority information not available
Research Initiative	X - not in an Initiative
Funding Scheme	X – not Funded via a specific Funding Scheme

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