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The role of NPR1 in Glutathione-mediated communication between pathogen resistance and excess light acclimation pathways in Arabidopsis
Reference
BBS/E/J/0000A125
Principal Investigator / Supervisor
Professor Andrew Maule
Co-Investigators /
Co-Supervisors
Institution
John Innes Centre
Department
John Innes Centre Department
Funding type
Research
Value (£)
50,793
Status
Completed
Type
Institute Project
Start date
01/08/2003
End date
31/07/2006
Duration
36 months
Abstract
Plants must coordinate gene expression in response to multiple stresses such as pathogen infection and exposure to excess light (EL). In the absence of salicylic acid (SA) signalling, NPR1, a key component of the SA pathway, suppresses foliar glutathione levels. This in turn determines how well leaves acclimate to EL. When SA signalling is activated, NPR1 suppression of glutathione level ceases. This inhibits acclimation making SA-induced leaves more sensitive to EL. Thus glutathione and NPR1 can mediate communication between pathogen resistance- and EL acclimation- pathways. This proposal will: 1) investigate the extent of glutathione-mediated regulation of the leaf transcriptome in response to combined stresses; 2) determine how NRP1-interacting proteins control glutathione levels; and 3) determine how glutathione can influence one or more signalling pathways.
Summary
unavailable
Committee
Closed Committee - Plant & Microbial Sciences (PMS)
Research Topics
X – not assigned to a current Research Topic
Research Priority
X – Research Priority information not available
Research Initiative
X - not in an Initiative
Funding Scheme
X – not Funded via a specific Funding Scheme
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