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Does the P110d isoform of the enzyme P13-K play a major role in leukaemia and lymphoma?

ReferenceBBS/E/B/0000H154
Principal Investigator / Supervisor Dr Martin Turner
Co-Investigators /
Co-Supervisors
Institution Babraham Institute
DepartmentBabraham Institute Department
Funding typeResearch
Value (£) 11,099
StatusCompleted
TypeInstitute Project
Start date 01/06/2005
End date 31/05/2006
Duration12 months

Abstract

Many cancers that arise in lymphocytes display significant changes in the mechanisms through which cells sense and respond to normal stimuli. Indeed, some lymphoid malignancies can be defined on the basis of the mechanisms which are dysfunctional. One important mechanism controlling malignancy is the P13-K enzyme system. We propose to test the hypothesis that the haematopoietic restricted P13-K isoform is a major contributor to lymphoid malignancy. If this is proven it paves the way for the use of specific modulators of this enzyme in malignancy.

Summary

unavailable
Committee Closed Committee - Biochemistry & Cell Biology (BCB)
Research TopicsX – not assigned to a current Research Topic
Research PriorityX – Research Priority information not available
Research Initiative X - not in an Initiative
Funding SchemeX – not Funded via a specific Funding Scheme
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