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The molecular mechanism of T4 phage activated proteolytic cleavage of Escherichia coli translation elongation factor Tu

ReferenceB07954
Principal Investigator / Supervisor Professor Colin Kleanthous
Co-Investigators /
Co-Supervisors
Institution University of East Anglia
DepartmentBiological Sciences
Funding typeResearch
Value (£) 144,943
StatusCompleted
TypeResearch Grant
Start date 13/10/1997
End date 13/10/2000
Duration36 months

Abstract

This proposal aims to dissect at the molecular level a defence mechanism that is activated in E. coli following infection by bacteriophage T4. A region of the T4 coat (the gol peptide) activates the Lit protease which cleaves EF-Tu leading to inhibition of translation. Three areas will be investigated: 1) The structure and mechanism of the Lit protease. 2) Mutagenesis of EF-Tu will probe the specificity of the cleavage reaction. The effects of nucleotide exchange and the cleavage of other GTP-binding proteins such as ras will also be analysed. 3) NMR will be used to determine the solution structure of the 29 residue gol peptide, which is sufficient to activate Lit. Spectroscopy and cross-linking will also be used to investigate the complexation of the gol peptide with either Lit or EF-Tu. The structure and activation properties of 'mutant' synthetic gol peptides will also be analysed.

Summary

unavailable
Committee Closed Committee - Biomolecular Sciences (BMS)
Research TopicsX – not assigned to a current Research Topic
Research PriorityX – Research Priority information not available
Research Initiative X - not in an Initiative
Funding SchemeX – not Funded via a specific Funding Scheme
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